학술논문

Loss of the batten disease protein CLN3 leads to mis-trafficking of M6PR and defective autophagic-lysosomal reformation.
Document Type
Article
Source
Nature Communications; 7/3/2023, Vol. 14 Issue 1, p1-19, 19p
Subject
REFORMATION
LYSOSOMAL storage diseases
PROTEINS
Language
ISSN
20411723
Abstract
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