학술논문
SEC is an antiangiogenic virulence factor that promotes Staphylococcus aureus endocarditis independent of superantigen activity.
Document Type
Article
Author
Source
Subject
*ASPARTATE aminotransferase
*STAPHYLOCOCCUS aureus
*HISTOCOMPATIBILITY class I antigens
*ENDOCARDITIS
RABBIT diseases
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*
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Language
ISSN
2375-2548
Abstract
The article presents a study which explores that Staphylococcus aureus endocarditis (SEC) as an antiangiogenic virulence factor that promotes Staphylococcus aureus endocarditis independent of superantigen activity. It mentions the SEC's ability to interfere with tissue revascularization and remodeling after injury serves as a mechanism to promote SAIE and its life-threatening systemic pathologies.