학술논문

Environmental Triggers of Acute Coronary Syndromes.
Document Type
Editorial
Source
Circulation. 6/14/2022, Vol. 145 Issue 24, p1761-1763. 3p.
Subject
*ACUTE coronary syndrome
*MYOCARDIAL infarction
*AIR pollution measurement
*TOBACCO smoke pollution
*CORONARY disease
*AIR pollution
*PARTICULATE matter
Language
ISSN
0009-7322
Abstract
Keywords: Editorials; acute coronary syndrome; air pollution; myocardial infarction; stress, physiological EN Editorials acute coronary syndrome air pollution myocardial infarction stress, physiological 1761 1763 3 06/10/22 20220614 NES 220614 B Article, see p 1749 b The term "acute coronary syndrome" comprises a range of presentations from unstable angina to myocardial infarction and sudden cardiac death, one that emerged as a direct consequence of improved understanding of the pathogenesis of atherothrombosis.[1] We now appreciate that coronary artery disease is a dynamic process involving plaque vulnerability, fibrinolytic function, and platelet activation responsible for the sudden transition from an apparently stable state to a potentially life-threatening acute event.[2] This understanding has culminated in effective and clinically meaningful personal and public health measures to prevent acute coronary syndromes. These associations were strongest for increases in air pollution levels in the hours immediately before symptom onset and were consistent in patients with unstable angina and myocardial infarction with or without ST-segment elevation. Editorials, acute coronary syndrome, air pollution, myocardial infarction, stress, physiological. [Extracted from the article]