학술논문
Noncanonical function of an autophagy protein prevents spontaneous Alzheimer's disease.
Document Type
Article
Author
Source
Subject
*AMYLOID plaque
*ALZHEIMER'S disease
*PHAGOCYTOSIS
*CENTRAL nervous system physiology
*TAU proteins
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Language
ISSN
2375-2548
Abstract
The article discusses the decline in expression of ATG16L in the brains of human patients with Alzheimer's disease (AD) suggests the possibility that a similar mechanism may contribute in human disease. It mentions that pharmacologic suppression of neuroinflammation reversed established memory impairment and markers of disease pathology in the AD model.